Definitely, ray parenchyma can divide after mechanical injury and localized death of the cambium. Often, that's where the callus pad originates that becomes the source of of new vascular cambium. This is well-described in standard texts.
In my own histology, I haven't seen involvement of proliferated cells associated with rays reach lenticels. I do not recall encountering published accounts of that either.
Actually, that is not surprising. Lenticels are in the phellem or cork. Phellem is derived from divsions of the phellogen (cork cambium). Most of those cells destined to be cork are pretty thick-walled and suberized. However, there are patches of thin-walled, less suberized (unsuberized?) parenchyma produced. Those patches become lenticels. As phellem matures, the living cells die. The thick-walled corky cells persist. The thin-walled cells break down, leaving air spaces. That's where the lenticels come from.
That's about as far as I can roll this. I'm thinking that no one else is interested in this thread by this point. I'll email you a paper, not so much on the 1st year anatomy above but on some of the consequences (Lenzian, K.J. 2006. J. Exp. Bot 57:2535-2546). I'm not attaching it because of copyright policies, but I'd email it to anyone else still interested!
Of course, I could be mistaken here. Standard texts can be incorrect as well, but I'm sort of curious why this is such an important point.